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This protective effect is achieved through the downregulation of GJ expression, suggesting that hepsin could be a viable therapeutic agent for APAP-induced hepatotoxicity

Increased cellular protein S -glutathionylation sensitized hepatocytes to TNF hepatotoxicity Increased cellular glutathionylated protein levels in response to GSSG accumulation prompted us to examine the effects of cellular protein S -glutathionylation on TNF-induced cell death

Nutr Hosp 43(3):428436

So if I do have a drink, I may risk increasing the inflammation and I don't want to do that

10.1016/j.crphar.2022.100084 230 PutmanS.AndersonF.MorrisonM