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Biomonitoring, exposure routes and risk assessment of chlorinated paraffins in humans: A mini-review
The dithiol mechanism was too slow to compete with the monothiol mechanism in the presence of GSH or GSSG and was only detected for the oxidation of roGFP2 using diamide-treated PfGrx without GSH
Ginkgetin at a concentration of 5 M caused accumulation of prostate cancer cells in the G0/G1 phase of the cell cycle in a time-dependent manner, 55.5% of cells were in the G0/G1 phase after 9 h treatment [38]
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