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In conclusion, the specific mechanism by which GLP-1RAs act on EAT remains elusive
Coronary and arterial plaque (atherosclerosis) develops gradually as cholesterol particles such as LDL in the blood are "lipid oxidized" and damage the blood vessels' lining, forming a plaque
This slow, controlled absorption supports consistent peptide delivery to the bloodstream, promoting more stable and sustained effects compared to injections or oral forms
All this means is we lack prospective trials spanning many decades and tens of thousands of patients
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