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Hepatic damage caused by alcoholism in some subjects may also result in synaptic dysfunction indirectly mediated by astrocytes, because increased ammonia levels that follow liver dysfunction diminish thrombospondin secretion by astrocytes and reduce the levels of synaptic proteins (Jayakumar et al., 2016)

When multiple publications originated from the same clinical research program, the most recent or most comprehensive report was used as the primary source

These advances similarly accelerate targeted therapeutic development

Semaglutide heralds significant advantages, and understanding its mechanism of action is crucial for optimal clinical utilization

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