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We conclude that oncogenic PIK3CA H1047R corrupts information transfer in a growth factor-dependent manner and suggest new opportunities for tuning of receptor-specific PI3K pathway outputs for therapeutic benefit

sin embargo, la naturaleza homeosttica (no proliferativa indiscriminada) y la ausencia de efectos mitgenos en tejidos sanos sugieren bajo riesgo terico

This Ca signaling influences neuronal activity and behavior by participating in bidirectional neuronglial communication

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Small extracellular vesicles and their miRNA cargo are anti-apoptotic members of the senescence-associated secretory phenotype