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attract immune and endothelial cells to injury sites
Copper may also be sequestered, along with deuterium, in the plaque deposits, under conditions of copper overload, which is toxic for the cell
The current picture is mechanistically credible and supported by small-scale human data, not clinically definitive
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In particular, the interaction of ROS with NO, which is the best characterized (and likely most important) mediator of endothelial function, is felt to have an important role in determining the causal relationship between oxidative stress and hypertension
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