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In addition, GLP-1R can be continuously activated to activate cAMP and P38 (94) and inhibit the conduction of the AKT and ERK1/2 signaling pathways (14, 16), ultimately inhibiting the proliferation of transplanted tumors in vivo and inducing the apoptosis of human pancreatic cancer cells in vitro ( 14)

These findings highlight the complex and potentially tissue-specific effects of GLP-1 RAs on cancer development and progression, underscoring the need for further research to elucidate the underlying mechanisms and clinical implications

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