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Silencing pannexin 1 (PANX1), an ATP release channel, reduced the expression of proinflammatory molecules, upregulated HO-1 expression, attenuated the MAPK/ERK pathway to alleviate NCOA4-mediated ferritinophagy, and ultimately inhibited ferroptosis and alleviated IRI-AKI (Fig

Additionally, the peptide might have reactivated the apoptosis pathway, a type of programmed cell death crucial for removing faulty cells, as evidenced by activity in caspases 3 and 7, which are enzymes that play key roles in apoptosis