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Individual results vary and depend on dose, duration and lifestyle changes alongside treatment

Loss of GPX4 protein expression in NRF1-deficient cells sensitized cells to ferroptosis, whereas in cells lacking GPX4, overexpression of NRF1 failed to inhibit ferroptosis

Emerging clinical evidence suggests that the SARS-CoV-2 virus can directly hijack or damage mitochondrial networks during acute infection, leaving a legacy of profound metabolic dysfunction long after the initial virus has been cleared