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Khng s dng trong trng hp sau (Chng ch nh) Khng s dng cho bnh nhn ung th tuyn gip hoc bnh nhn c cc triu chng lin quan n khi u tuyn gip
Neurology 1998;50:1934
The anti-inflammatory pathways modulated by GLP-1 receptor activation (NF-kB suppression, macrophage polarization, Treg enhancement) are largely distinct from the pathways targeted by: Methotrexate: Folate metabolism disruption, different anti-inflammatory pathway TNF inhibitors (adalimumab, etanercept): Direct TNF blockade, whereas GLP-1 reduces TNF production upstream IL-6 inhibitors (tocilizumab): Receptor-level blockade, while GLP-1 reduces IL-6 production JAK inhibitors (tofacitinib, baricitinib): Different signaling pathway entirely Corticosteroids: Broad immunosuppression, while GLP-1 is immunomodulatory This pathway complementarity is why additive benefit is biologically plausible
Sometimes slower
Over time, it can cause irreversible nerve damage, cognitive decline, and mental health disorders such as depression
