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HIF-1 activates glycolysis-related genes, including pyruvate dehydrogenase kinase 1 (PDK1), increasing the conversion of glucose to pyruvate and subsequently to lactate by inactivating PDH, which is considered critical for metabolic adaptation to hypoxia [74] [Figure 1F]

Administration: Intravenous (IV) or intramuscular (IM) injection

American Journal of Hypertension

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Inhibition of PPAR using a specific antagonist (GW9662) reverses the effects of OCSCs, restoring NF-B activity and promoting M1 polarization