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BPC-157 originates from gastric juice proteins and works primarily through nitric oxide system modulation, VEGF/EGF/FGF upregulation, and FAK-paxillin pathway activation with the strongest research support for gut and tendon healing
L-carnitine is used for replacement therapy
Furthermore, tubular-specific knockout of pyruvate kinase M2 (PKM2), a key enzyme in glycolysis, has been reported to protect ischemic AKI by switching the metabolism to PPP (Zhou et al., 2019)
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