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Acylcarnitine is released from abnormal mitochondria in peripheral glial cells to affect surrounding axons, resulting in the destruction of axonal membrane properties and the induction of axonal degeneration, which has been shown to be neurotoxic [116]
Long-term safety data remains limited for most of these compounds, so risks beyond short trials or animal studies are largely unknown
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If budgeting for one intervention, prioritise tirzepatide it has FDA approval and robust Phase 3 evidence for weight loss, whereas glutathione supplementation for metabolic support does not
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