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A competing-risks regression model identified several independent predictors of treatment-emergent obesity (TAF/FTC+DTG, baseline CD4 count, baseline viral load, and baseline body mass index) and a 10% or greater weight gain (TAF/FTC+DTG, baseline CD4 count, baseline viral load, baseline weight, female sex, and age)
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(Endocrinology, 2001) demonstrated in obese rodent models that AOD-9604 stimulated fat breakdown and inhibited lipogenesis without producing measurable changes in IGF-1 concentrations or causing the glucose intolerance associated with full-length GH administration [6]
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