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Evaluating these trial metrics through a biomechanical and neurotologic lens clarifies the precise clinical role for SPI-1005: It acts as a selective metabolic protector for auditory tissue hardware, yet it remains incapable of resolving mechanical endolymphatic pressure shifts or underlying sensory integration deficits
Notably, its half-life has been reported to be around 4 h in rats (Sturis et al., 2003), suggesting that it may not act as long in animals as it does in humans
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