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The dual mechanism produces additive or combined effects on energy balance -- GIPR activation in the CNS may enhance the anorectic signals from GLP-1R activation, and peripheral GIPR signaling improves adipose tissue function and lipid handling in ways GLP-1 alone does not

To substantiate this hypothesised mechanism for the effect of GLP-1 in the OB of WD mice, we performed OGTT to test whether prior IP injection of an 2 -adrenergic receptor agonist, UK14304 23 , could modulate the effect of bulbar Ex4 administration (Fig

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