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Deficiency in the level of activity of glucose-6-phosphate dehydrogenase (G6PD) is the basis of favism, primaquine (an anti-malarial drug) sensitivity and several other drug-sensitive hemolytic anemias, anemia and jaundice in the newborn, and chronic nonspherocytic hemolytic anemia

128 These insights support the integration of immune-restorative strategies, including checkpoint inhibitors, innate immune activators or adoptive cell therapies, into standard treatment paradigms

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This system exhibits high specificity for cystine and glutamate, facilitating the uptake of cystine into cells while exporting glutamate extracellularly

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